rabbit polyclonal anti bid (Cell Signaling Technology Inc)
Cell Signaling Technology Inc is a verified supplier 
Cell Signaling Technology Inc manufactures this product 
96
Structured Review
Cell Signaling Technology Inc
rabbit polyclonal anti bid
Rabbit Polyclonal Anti Bid, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1484 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti-bid/BID+Antibody/pmc12182575-1-0-6
Average 96 stars, based on 1484 article reviews
Rabbit Polyclonal Anti Bid, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1484 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti-bid/BID+Antibody/pmc12182575-1-0-6
Average 96 stars, based on 1484 article reviews
rabbit polyclonal anti bid - by Bioz Stars,
2026-09
96/100 stars
Images
Related Articles
Cell Culture:Article Title: Antagonistic nanobodies implicate mechanism of GSDMD pore formation and potential therapeutic application Article Snippet: Not possible in cell culture experiments that were immediately measured by flow cytometry of ( Article Title: Preferential target is mitochondria in α-mangostin-induced apoptosis in human leukemia HL60 cells Article Snippet: Our previous study has shown that a-mangostin, a xanthone from the pericarps of mangosteen, induces caspase-3dependent apoptosis in HL60 cells.. In the current study, we investigated the mechanism of apoptosis induced by a-mangostin in HL60 cells. a-Mangostin-treated HL60 cells demonstrated caspase-9 and -3 activation but not -8, which leads us to assume that a-mangostin may mediate the mitochondrial pathway in the apoptosis.. Parameters of mitochondrial dysfunction including swelling, loss of membrane potential (DWm), decrease in intracellular ATP, ROS accumulation, and cytochrome c/AIF release, were observed within 1 or 2h after the treatment. Flow Cytometry:Article Title: Antagonistic nanobodies implicate mechanism of GSDMD pore formation and potential therapeutic application Article Snippet: Not possible in cell culture experiments that were immediately measured by flow cytometry of ( Article Title: Preferential target is mitochondria in α-mangostin-induced apoptosis in human leukemia HL60 cells Article Snippet: Our previous study has shown that a-mangostin, a xanthone from the pericarps of mangosteen, induces caspase-3dependent apoptosis in HL60 cells.. In the current study, we investigated the mechanism of apoptosis induced by a-mangostin in HL60 cells. a-Mangostin-treated HL60 cells demonstrated caspase-9 and -3 activation but not -8, which leads us to assume that a-mangostin may mediate the mitochondrial pathway in the apoptosis.. Parameters of mitochondrial dysfunction including swelling, loss of membrane potential (DWm), decrease in intracellular ATP, ROS accumulation, and cytochrome c/AIF release, were observed within 1 or 2h after the treatment. Fluorescence:Article Title: Antagonistic nanobodies implicate mechanism of GSDMD pore formation and potential therapeutic application Article Snippet: Not possible in cell culture experiments that were immediately measured by flow cytometry of ( Article Title: Preferential target is mitochondria in α-mangostin-induced apoptosis in human leukemia HL60 cells Article Snippet: Our previous study has shown that a-mangostin, a xanthone from the pericarps of mangosteen, induces caspase-3dependent apoptosis in HL60 cells.. In the current study, we investigated the mechanism of apoptosis induced by a-mangostin in HL60 cells. a-Mangostin-treated HL60 cells demonstrated caspase-9 and -3 activation but not -8, which leads us to assume that a-mangostin may mediate the mitochondrial pathway in the apoptosis.. Parameters of mitochondrial dysfunction including swelling, loss of membrane potential (DWm), decrease in intracellular ATP, ROS accumulation, and cytochrome c/AIF release, were observed within 1 or 2h after the treatment. Microscopy:Article Title: Antagonistic nanobodies implicate mechanism of GSDMD pore formation and potential therapeutic application Article Snippet: Not possible in cell culture experiments that were immediately measured by flow cytometry of ( Article Title: Preferential target is mitochondria in α-mangostin-induced apoptosis in human leukemia HL60 cells Article Snippet: Our previous study has shown that a-mangostin, a xanthone from the pericarps of mangosteen, induces caspase-3dependent apoptosis in HL60 cells.. In the current study, we investigated the mechanism of apoptosis induced by a-mangostin in HL60 cells. a-Mangostin-treated HL60 cells demonstrated caspase-9 and -3 activation but not -8, which leads us to assume that a-mangostin may mediate the mitochondrial pathway in the apoptosis.. Parameters of mitochondrial dysfunction including swelling, loss of membrane potential (DWm), decrease in intracellular ATP, ROS accumulation, and cytochrome c/AIF release, were observed within 1 or 2h after the treatment. Biomarker Discovery:Article Title: Antagonistic nanobodies implicate mechanism of GSDMD pore formation and potential therapeutic application Article Snippet: Not possible in cell culture experiments that were immediately measured by flow cytometry of ( Article Title: Preferential target is mitochondria in α-mangostin-induced apoptosis in human leukemia HL60 cells Article Snippet: Our previous study has shown that a-mangostin, a xanthone from the pericarps of mangosteen, induces caspase-3dependent apoptosis in HL60 cells.. In the current study, we investigated the mechanism of apoptosis induced by a-mangostin in HL60 cells. a-Mangostin-treated HL60 cells demonstrated caspase-9 and -3 activation but not -8, which leads us to assume that a-mangostin may mediate the mitochondrial pathway in the apoptosis.. Parameters of mitochondrial dysfunction including swelling, loss of membrane potential (DWm), decrease in intracellular ATP, ROS accumulation, and cytochrome c/AIF release, were observed within 1 or 2h after the treatment. |
